Dangers of Hartz Flea — Tick Shampoo, Animals

Dangers of Hartz Flea & Tick Shampoo

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dog image by Michal Tudek from Fotolia.com

A product of a Secaucus, NJ based veterinary company, Hartz Flea & Tick Shampoo is a shampoo that is used on cats and dogs to treat flea and tick infestations. If left untreated, fleas and ticks can cause a host of problems to the dog or cat, such as flea allergy, tapeworm, dermatitis and flea anemia. However, some users of Hartz Flea and Tick shampoo have reported various adverse reactions to the product.

Poisioning

Some users have complained that this product, when used on cats and dogs, almost had the effect of killing not the fleas and ticks, but the host animal itself. According to anecdotal reports, animals treated with Hartz Flea and Tick have started showing signs of neurological issues such as epilepsy and hallucination. These were accompanied by shock, pain, swelling in the eyes and reddening of the belly. Hartz employs several potentially toxic chemicals as the active ingredients, leading to this potentially dangerous reaction. A class action lawsuit was filed regarding these reported claims, but no definitive statements have been made by a medical authority regarding the truth or falsity of these claims.

Hair Loss

Some owners have reported that pets lose some or all hair after an application of Hartz flea and tick shampoo. Many of these animals experienced itchiness before the hair loss occurred. The hair loss has not been conclusively linked to Hartz Flea and Tick Shampoo, but it occurred shortly after an application of the shampoo in several cases. In most instances, the hair eventually regrows.

Seizures and Other Neurological Problems

Some animals that were administered Hartz Flea & Tick Shampoo also experienced other cerebral dysfunctions including seizures. This occurred almost immediately after the use of the product. Other pet owners report sudden debilitation or coma after the use of Hartz Flea and Tick products. These adverse side effects are attributed to the presence in the product of organophosphate insecticides (OPs) and carbamates. Ingredients used in Hartz that pose dangers include diazinon, chlorpyrifon, naled, phosmet, dichlorvos, malathion and tetrachlorvinphos.

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Cat and dog flea treatments can be toxic to pets, humans

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CBC Marketplace investigates risks to humans and pets from popular pet products

CBC Marketplace has discovered that more than 2,000 animals are reported to have died in North America since 2008 as a result of exposure to flea and tick treatment products, which can contain dangerous chemicals that kill fleas but can also harm pets.

Some researchers are also concerned that pesticide exposures from flea treatments could have consequences for humans, especially small children.

“It’s one of those things that is incredibly unfair — it’s unfair for families, it’s unfair for their pets, it’s unfair for kids. And the truth is, there are better options,” public health scientist Miriam Rotkin-Ellman told Marketplace co-host Erica Johnson.

“We don’t need to put our kids at risk, don’t need to put our families at risk, our pets at risk.”

According to information Marketplace obtained from the Pest Management Regulatory Agency (PMRA), Health Canada received 4,726 incident reports for cats and dogs related to topical flea treatments between 2009 and 2013.

Health Canada receives reports about incidents related to Canadian products used domestically and in the United States. Almost two thirds (62 per cent) of these reports were for animals in Canada.

The deaths included 1,188 cats and 872 dogs, most of them in the United States.

Flea treatments can include collars, sprays, powder, shampoos and “spot-on” treatments, where pet owners dab a small amount of chemical directly onto the animal’s fur. Some of the treatments contain pesticides that target the nervous systems of fleas and ticks.

Spot-on treatments are responsible for approximately 80 per cent of the incidents, according to the PMRA. Most of the reports involve over-the-counter treatments.

For tips on keeping your pet safe, follow the conversation with @cbcmarketplace on Twitter, using the hashtag #pets.

Marketplace also investigated how pesticides in flea collars can transfer from the collar to elsewhere in the home, which could raise special concerns for families with small children.

“There’s mounting evidence that pesticides can be really harmful for kids at low levels,” says Rotkin-Ellman.

“When pesticides are on pets, they come into contact with kids all the time.”

The investigation, “Paws for Concern,” airs Friday, Dec. 5 at 8 p.m. (8:30 p.m. NT) on CBC Television. The show also tested dog harnesses, evaluating popular brands on the market to see if they actually work in an accident.

Dangers to small pets

Pet owners need to be extremely careful in how they choose flea treatments and how they use them on their pets, says Dr. Whitney Chin, a London, Ont.-based veterinarian.

Dr. Chin says one chemical common in over-the-counter dog treatments — permethrin — is of particular concern. Using too much of the chemical can be dangerous, and it is highly toxic to cats.

“I’ve seen it far too many times, unfortunately,” Dr. Chin says.

“Some people think, A cat is a small dog and I’ll just use a little dose,” Dr. Chin says.

Cats can also be inadvertently exposed if a dog in the home is treated with a product and “the dog or cat socialize, share the same bedding or if the cat grooms the dog.”

Chemicals transferred in this way can be enough to seriously harm a cat, he says.

According to the website of the Canadian Veterinary Medical Association (CVMA), “Toxicity [to cats] from dog flea and tick products is a medical emergency.”

Symptoms of exposure can include uncontrollable shaking, and the CVMA advises taking affected animals to a vet immediately. “The longer your cat is left to shake, the greater the chance of permanent damage (death included).”

Dr. Chin advises talking to your veterinarian before choosing a treatment product, and says that not all products on the market are dangerous.

New labelling rules

New labelling rules came into effect in Canada in 2012, requiring some treatments for dogs to show a clear warning label that the product can be toxic to cats.

However, products manufactured before the new rules were introduced were not recalled and may still be on the market.

Marketplace found treatments on store shelves in fall 2014 that did not have the new labels. Health Canada says it will investigate these cases.

Dr. Chin is concerned that the new warning labels do not go far enough and should more prominently alert pet owners to the dangers of poisoning.

Pet store staff may not always tell people who have both dogs and cats about the risks of exposure to both animals.

“People should be aware,” Dr. Chin says.

Human health concerns

Some researchers are concerned that pesticides in flea collars can also pose a risk to people, even when used properly.

Rotkin-Ellman at the U.S. Natural Resources Defense Council tested pets to see how much pesticide from flea collars the rest of us may be exposed to.

“Flea collars are designed to release a toxic substance that kills fleas on the pet’s fur,” she says. However, she says, “it also can get on the bedding, it can get on kids’ hands, it can go all sorts of places.”

See also:  How To Get Rid Of Raccoons Permanently

Her team tested pesticide residues left on fur after monitoring a pet that wore a flea collar for three days.

“We found much higher levels than we expected,” she says.

The team found that kids could be exposed to higher levels than are considered safe by the Environmental Protection Agency. This was a particular concern for small children, who could be exposed to chemicals absorbed through their skin, or if they put their hands in their mouths.

Rotkin-Ellman says that exposure to the types of pesticides used in flea collars may be linked to behavioural problems, cognitive delay and problems with motor development.

She advises people who are concerned about pesticide exposure that there are other ways to control fleas, including bathing your pet and washing bedding regularly.

www.cbc.ca

The Truth About Coronavirus In Humans And Pets

By Matthew Russell

The World Health Organization (WHO) has declared the novel coronavirus, also called COVID-19, a public health emergency of international concern. This is a rarely given designation from the WHO, and reason for the global population to take note. COVID-19 is a serious health hazard, able to pass between people by way of tiny water droplets in the air we breathe.

If you are concerned about your own health and the health of your pet, you are not alone. However, there may be less to worry about than you think. Staying abreast of the facts and taking appropriate precautions will help you stay safe and feel more at ease.

Source: Pexels
Coronavirus disease has sent thousands to the hospital with potentially fatal symptoms.

WHAT IS CORONAVIRUS

The name “coronavirus” was first given to the disease when scientists discovered its shape under an electron microscope. The virus appears as a ring, circled by small “ornaments,” much like a coronet or crown.

Coronaviruses are a family of viruses that have been around for a long time. Some strains of this virus are common in animals, and in rare occurrences, some strains are able to mutate in order to be able to infect humans through contaminated meat, leading to epidemics like the MERS (Middle East Respiratory Syndrome) and SARS (Severe Acute Respiratory Syndrome) outbreaks. COVID-19 is a completely new strain of the virus, which has its own separate set of characteristics.

COVID-19 is a virus that causes fever and respiratory issues such as trouble breathing and a dry cough. The symptoms are mild or nonexistent for most people, but the elderly and those with compromised immune systems and underlying health conditions may experience more serious symptoms and can even die.

Source: Pexels
The disease is thought to have originated in a fish and animal market in Wuhan, China.

WHERE DID CORONAVIRUS DISEASE COME FROM?

We still aren’t sure where COVID-19 originated, though it’s been linked to unsanitary conditions in the global wildlife trade, much of which originates from the east coast of China. Only 27 of the first 41 people affected by the disease had visited the animal and fish market in Wuhan, the Lancet reports.

What’s important to note, however, is that this danger is most commonly present in live-animal markets, particularly those where freshly butchered meats from different animals are stacked on top of one another, allowing their blood and viruses to mingle and potentially form newly mutated virus strains capable of infecting humans. There is no evidence at this time that you can contract COVID-19 from a pet animal.

Source: Pexels
The disease is passed through saliva or tiny water droplets in the air we breathe.

HOW IS CORONAVIRUS DISEASE PASSED AROUND?

More than 80,000 cases of COVID-19 have been documented in China since the outbreak began in December 2018. Containment of the disease has been imperative but easier said than done. In the last several weeks, the virus has traveled around the globe, infecting over 220,000 people and killing nearly 10,000 to date.

Researchers at Imperial College London maintain that the average COVID-19-infected individual will pass the virus on to at least two-and-a-half others. In contrast, someone infected with the measles is likely to pass the disease on to 12 to 18 others.

However, one place you’re not going to get COVID-19 from is your pet. Dogs are capable of being infected with a strain of coronavirus called canine coronavirus (CCoV), but they are not able to transmit COVID-19.

“While there has been one instance of a dog being infected in Hong Kong, to date,” the WHO reports, “there is no evidence that a dog, cat or any pet can transmit COVID-19. COVID-19 is mainly spread through droplets produced when an infected person coughs, sneezes, or speaks. To protect yourself, clean your hands frequently and thoroughly.”

Source: Pexels
In kitten, coronavirus disease can lead to death about 5 to 10% of the time.

ARE PETS IN DANGER OF CONTRACTING CORONAVIRUS DISEASE?

Your pet is not in danger of getting the COVID-19 virus. He or she may be able to catch CCoV if exposed to it, but it is not the same as the COVID-19 virus that currently has the world on alert.

“Many animals are vulnerable to coronavirus, the Mercury News reports, “but it’s not the scary one. And they can’t give it you.”

Source: Pexels
So far, pets have not contracted the new coronavirus disease, nor passed it on to their humans.

So no. We have no reason to believe you will pass COVID-19 to your pet if you get it, and your pet is unable to transmit the disease to you either. It’s one of the many benefits of having friends that are not the same species as you! So if you’re in quarantine or practicing social distancing (nice work, responsible human!), take the extra free time you have to enjoy your amazing pet without fear that COVID-19 might pass between the two of you.

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Dog tick — danger to people and animals

Dermacentor variabilis (Say), also known as the American dog tick or wood tick, is found predominantly in the United States, east of the Rocky Mountains, and as its name suggests, is most commonly found on dogs as an adult. The tick also occurs in certain areas of Canada, Mexico and the Pacific Northwest of the U.S. (Mcnemee et al. 2003). Dermacentor variabilis is a 3-host tick, targeting smaller mammals as a larva and nymph and larger mammals as an adult. Although it is normally found on dogs, this tick will readily attack larger animals, such as cattle, horses, and even humans. The 8-legged adult is a vector of the pathogens causing Rocky Mountain spotted fever (RMSF) and tularemia, and can cause canine tick paralysis. While the American dog tick can be managed without pesticides, when necessary a recommended acaricide is an effective way of eliminating an existing tick infestation near residences.

Figure 1. Adult female American dog tick, Dermacentor variabilis (Say). Photograph by Lyle J. Buss, University of Florida.

Distribution (Back to Top)

The American dog tick is widely distributed in the United States east of a line drawn from Montana to South Texas. It is also found in Canada, east of Saskatchewan, and in California, west of the Cascade and the Sierra Nevada Mountain ranges. This species is most abundant in the eastern United States from Massachusetts south to Florida but is also common in more central areas of the U.S., including Iowa and Minnesota (Matheson 1950).

It was been suggested that adult ticks move to the edge of the roads and trails in an attempt to find a host, or «quest.» Some have hypothesized that because many animals typically follow trails, they leave an odor that attracts these ticks causing them to move toward and quest alongside trails in attempts to find a host (Mcnemee et al. 2003).

Description (Back to Top)

The 8-legged adult male and female D. variabilis ticks are typically brown to reddish-brown in color with gray/silver markings on their scutum (dorsal «shield»). The female will vary in size depending on whether or not it has blood fed. Unfed females are typically 5 mm long and are slightly larger than males, which are about 3.6 mm long. Females can be distinguished by a short or small dorsal scutum, right behind the mouthparts while the male scutum covers the majority of its dorsal surface. Blood-fed (engorged) females can enlarge up to 15 mm long and 10 mm wide.

Figure 2. Dorsal view of adult female American dog tick, Dermacentor variabilis (Say), with body parts marked. Photograph by J.F. Butler, University of Florida.

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Figure 3. Dorsal view of American dog ticks, Dermacentor variabilis (Say), with male on left, and female on right. Photograph by J.F. Butler, University of Florida.

Figure 4. Ventral view of American dog ticks, Dermacentor variabilis (Say), with male on left, and female on right. Photograph by K. Wilson, University of Florida.

Figure 5. Engorged adult female American dog tick, Dermacentor variabilis (Say). Photograph by K. Wilson, University of Florida.

The mouthparts and their base, also known as the capitulum, are visible when viewing from above, with the second segment of the palps about as long as it is wide (Smith and Whitman 1992). The anal and genital openings occur on the underside of ticks. Only adults possess genital openings. Posterior to the anus, there is a groove and the spiracular plate is directly behind the fourth coxae (leg attachment segment).

Figure 6. Dorsal view of the head region from the American dog tick, Dermacentor variabilis (Say). For an even closer look at the mouthparts at the left of the image, seen slightly lower than a line drawn through the center of the image, click here. Photograph by Janice Carr, Center for Disease Control.

There are two stages of immature ticks: 6-legged larvae and 8-legged nymphs. Larvae are

0.62 mm long and are yellow before blood-feeding and gray to black when engorged. Nymphs are about 0.9 mm long, and a pale, yellow-like brown before blood-feeding and become slate gray when engorged (Smith and Whitman 1992). Both stages of immature tick will have red markings near their eyes and will lack white coloration on the scutum (shield). Nymphs can be distinguished from adults by the lack of a genital opening.

Figure 7. Relative sizes of American dog ticks, Dermacentor variabilis (Say), with male (left), female (center), nymph (right). Photograph by K. Wilson, University of Florida.

Life Cycle (Back to Top)

Dermacentor variabilis develops from the egg stage, to the 6-legged larva, to the 8-legged nymph, and finally to the adult. The cycle requires a blood meal before progression from larva to nymph, from nymph to adult and by the adult for egg production. This cycle also requires three different hosts and requires at least 54 days to complete, but can take up to two years depending on the host availability, host location and the temperature.

Figure 8. Life cycle of the American dog tick, Dermacentor variabilis (Say). Photograph by Center for Disease Control.

After five to 14 days of blood feeding, a fully engorged female D. variabilis drops from the host. She digests the blood meal and develops her egg clutch over the next four to 10 days. She then lays anywhere from 4,000 to 6,500 eggs on the ground (Matheson 1950). About 26 to 40 days later, depending on the temperature, the eggs hatch into larvae (James et al. 1969).

After hatching, larvae remain on the ground or climb growing vegetation where they wait for small mammals, such as mice, to serve as hosts for their first blood meal. This host location behavior is called questing. Under favorable conditions, larvae can survive up to 11 months without feeding. After contacting and attaching to a host, larvae require from two to 14 days to complete blood feeding. After feeding, larvae detach from their host and fall to the ground where they digest their blood meal and molt into the nymphal stage. This process can take as little as a week, although this period is often prolonged.

Nymphs can survive six months without a blood meal. After successfully questing for their second host, which is normally a slightly larger mammal (such as a raccoon or opossum), the nymphs will blood feed over a three to 10-day period. After engorging, they fall off the host, digest their blood meal and molt into an adult. This process can take anywhere from three weeks to several months.

Adults can survive two years without feeding, but readily feed on dogs or other larger animals when available. Questing adult ticks climb onto a grass blade or other low vegetation, cling to it with their third pair of legs, and wave its legs as a potential host approaches. As the hosts brush the vegetation, the ticks grab onto the passing animal. Mating occurs on the host and the female engorges within six to 13 days after which she drops from the host to lay her eggs and then she dies, thus completing the cycle (Matheson 1950).

Seasonality (Back to Top)

Adult American dog ticks overwinter in the soil and are most active from around mid-April to early September. Larvae are active from about March through July and nymphs are usually found from June to early September (Goddard 1996). In northern areas, such as Massachusetts and Nova Scotia, adults appear from April to August with a peak in May and June (Campbell 1979, McEnroe 1979a). In central latitudes of the U.S., such as Virginia, adults are found to be active from April to September/October with peaks in May and July (Sonenshine and Sout 1971, Carroll and Nichols 1986). In Ohio, adult activity occurred between April and September with a peak in May/June and a second smaller peak in August/September (Conlon and Rockett 1982).

A study done in Lexington, Kentucky, found the duration of D. variabilis‘ spring activity was related to its overwintering success. This study also concluded that overwintering adult D. variabilis ticks remained active throughout the entire season (Burg 2001). This same study reported that adult activity began a week or two earlier than in more northern states, such as Ohio, and a week or two later than in more southern states, such as Georgia. In Georgia, adult ticks are active from late March to August with peaks from early May to late June (Newhouse 1983). While in Florida, adult D. variabilis activity occurs from April to July (McEnroe 1979b).

Medical and Veterinary Importance (Back to Top)

The American dog tick is the primary vector for the pathogen causing Rocky Mountain spotted fever (RMSF), although it is also known to transmit the causative agent of tularemia and can cause canine tick paralysis. First discovered in the late 19th century in the Rocky Mountain region, RMSF is more commonly reported in eastern U.S. Rickettsia rickettsii, the causative agent of RMSF, is primarily vectored by D. variabilis to dogs and humans following its acquisition from rodents. Overwintering larvae can also acquire RMSF transovarially (mother to egg) yielding RMSF-infected larvae (Piesman and Gage 1996). Because larval and nymphal ticks rarely bite humans, the adult tick is the primary lifestage of concern for humans. In the southeast U.S., peak occurrence of the disease is in July, at or shortly after the peak activity of adult D. variabilis.

Rocky Mountain spotted fever is an infectious disease. The rickettsia affect small peripheral blood vessels causing rashes to develop within two to five days. These rashes are known to start in the wrists and ankles and move up to the rest of the body. Because of its direct association with adult D. variabilis, RMSF is a seasonal disease, and is more prevalent during the months between April and September. Symptoms usually appear within two to 14 days, with averages of around a week. Fever, nausea, vomiting, diarrhea, headaches, muscle pain and lack or appetite are all symptoms of this disease. Clinical symptoms include elevated liver enzymes, abnormal platelet count, and electrolyte abnormalities (ALDF 2006). Mortality from this disease in humans is 20 to 25% if untreated and 5% with appropriate clinical therapy. In order for transmission to occur, however, the tick must be attached for six to eight hours and in some cases transmission requires more than 24 hours (Thorner et al. 1998).

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Dermacentor variabilis can also vector Francisella tularensis, the organism causing tularemia. This gram negative coccobacillus (bacteria) also can be transmitted by contact with arthropods including other ticks, deer flies, mosquitoes, fleas, as well as from infected animals (principally rabbits) to hunters and ranchers. However person-to-person contact is rare. Incubation for this disease is usually three to five days but can take up to 21 days for symptoms to appear (CCHDOE 2001). Symptoms of tularemia include chills, fever, prostration, ulceration at the site of the bite, and tender, swollen lymph nodes. If untreated, fatality rate may be as high as 5 to 7%. Tularemia occurs only in the northern hemisphere and most frequently in Scandinavia, North America, Japan, and Russia (Ellis et al. 2002).

Canine tick paralysis can occur due to the feeding of D. variabilis. In this case, the tick will attach to the back of the dog’s neck, or at the base of the skull, and feed for at least five to six days. It is believed to release a salivary gland protein into the body. Paralytic symptoms then become visible through unsteadiness and loss of reflex actions. If the tick is not removed, respiratory failures can be fatal. Such paralysis is not limited to dogs, as it can happen to children as well. Once the tick is properly removed, recovery is usually within one to three days (Schmitt 1969). In the U.S., the fatality rate is about 10% in the Pacific Northwest, and most of those who die are children (Gregson 1973).

Removal (Back to Top)

Ticks should not be removed by handpicking because infected tick secretions can be transferred from a person’s hands to his or her eyes, mucous membranes, mouth, etc. Therefore, forceps should be used when removing a tick (Smith and Whitman 1992). To properly remove a tick, grasp the mouthparts near the attachment site firmly with tweezers. Once the mouthparts are held, the tick should be pulled straight back slowly to ensure the entire mouthparts are removed from the body. It is important that the tick is removed slowly because the mouthparts are covered in sharp, backward directed barbs which assist the tick in holding onto its host (Parsons et al. 1989). Sometimes, by removing the tick, a piece of the hosts skin may break off, which should not be a great concern, but bears watching for infection or later reaction.

Figure 9. Enlarged view of the mouthparts of an American dog tick, Dermacentor variabilis (Say). Notice the hundreds of sharp, backward-directed barbs which assist the tick in holding onto its host. For an even closer look at these barbs, click here. Photograph by Janice Carr, Center for Disease Control.

Figure 10. Enlarged view of the hundreds of sharp, backward-directed barbs on the mouthparts of an American dog tick, Dermacentor variabilis (Say). Photograph by Janice Carr, Center for Disease Control.

To treat the wound, it is important to wash the area with soap and water followed by an antiseptic. Once the tick is removed, the recommendation is to save it in alcohol, with the date the tick was removed, in case it is needed for identification or pathogen testing, should disease symptoms occur. If no symptoms occur within a month of removal, the tick may be discarded (USACHPPM 2003).

Management (Back to Top)

The American dog tick occurs primarily in wooded, shrubby and long-grass areas. However, it is possible for residential areas to support populations of this tick. Shrubs, weeds, tall grass, clutter and debris on the property attracts the rodents that are hosts for immature ticks. By maintaining grass short, removing possible rodent harborages, and sealing cracks and crevices in and around the property one can directly reduce or prevent local tick populations. Keeping grass and weeds cut short decreases humidity, which helps kill ticks or makes an area undesirable for ticks and rodents. Additionally, it makes it difficult for ticks to climb on the vegetation and wait for its host. If pesticides are applied, cutting the vegetation short increases effectiveness and allows for better coverage. Removing rodent harborage areas may reduce an infestation.

Because dogs can easily pick up ticks while walking on infested grass or roaming through wooded areas, it is necessary to treat the pet properly. There are many products that can be applied to prevent or treat a tick infestation on an animal including topical treatments and sprays. Regularly grooming, washing bedding, and examining the dog are strongly recommended to prevent tick infestations.

Selected References (Back to Top)

  • American Lyme Disease Foundation (ALDF). (2006). Rocky Mountain spotted fever. http://www.aldf.com/RMSF.shtml (23 September 2008).
  • Burg JG. 2001. Seasonal Activity and spatial distribution of host-seeking adults of the tick Dermacentor variabilis. Medical and Veterinary Entomology 15: 413-421.
  • Campbel A. 1979. Ecology of the American dog tick, Dermacentor variabilis in southwestern Nova Scotia. Recent Advances in Acarology. Rodriguez JG (editor). Volume 2: 135-143.
  • Carroll JF, Nichols JD. 1986. Parasitization of meadow voles, Microtus pennsylvanicus (Ord), by American dog ticks, Dermacentor variabilis (Say), and adult tick movement during high host density. Journal of Entomological Science 21: 102-113.
  • Clark County Health District Office of Epidemiology (CCHDOE). (2001, May 11). Tularemia. http://www.southernnevadahealthdistrict.org/physician/download/physician_tularemia.pdf (23 September 2008).
  • Conlon JM, Rockett CL. 1982. Ecological investigations of the American dog tick, Dermacentor variabilis (Say), in northwest Ohio (Acari: Ixodidae). International Journal of Acarology 8: 125-131.
  • Goddard J. 1996. Physician’s Guide to Arthropods of Medical Importance. pp. 287-302. CRC Press. Jackson, Mississippi.
  • Gregson JD. 1973. Tick paralysis: an appraisal of natural and experimental data. Monograph No. 9. Canada Department of Agriculture, Ottawa.
  • Eddis J, Oyston CF, Green M, Titball RW. 2002. Tularemia. Clinical Microbiology Reviews: 15: 631-646.
  • James M, Robert FH. 1969. Herm’s Medical Entomology. 6th Edition. pp. 326-329. The Macmillan Company. Toronto, Ontario.
  • Koehler PG, Oi F. (2003). Ticks. EDIS. http://edis.ifas.ufl.edu/IG088 (23 September 2008).
  • Matheson R. 1950. Medical Entomology, 2nd Edition. Comstock Publishing Company, Inc. Ithaca, NY.
  • McEnroe WD. 1979a. Dermacentor variabilis (Say) in eastern Massachusetts. Recent Advances in Acarology. Rodriguez JG (editor). Volume 2: 145-153.
  • McEnroe, W.D. 1979b. The effect of the temperature regime on Dermacentor variabilis (Say) populations in eastern North America. Acarologia 20: 58-67.
  • Mcnemee RB, Sames WJ, Maloney Jr FA. 2003. Occurrence of Dermacentor variabilis (Acari:Ixodidae) around a porcupine (Rodentia: Erthethizontidae) carcass at Camp Ripley, Minnesota. Journal of Medical Entomology 40: 108-111.
  • Newhouse VF. (1983) Variations in population density, movement, and rickettsial infection rates in a local population of Dermacentor variabilis (Acarina: Ixodidae) ticks in the piedmont of Georgia. Environmental Entomology 12: 1737-1746.
  • Parsons GL, Rossignol PA. 1989. Identifying adult hard ticks commonly found on humans in Oregon. EM8410. Oregon State University Extension Service.
  • Piesman J, Gage KL. 1996. Ticks and mites and the agents they transmit. The Biology of Disease Vectors. Beaty BJ, Marquardt WC (editors). pp. 160-174. University Press of Colorado. Newot, CO.
  • Schmitt N. Bowner EJ, Gregson JD. 1969. Tick paralysis in British Columbia. Canadian Medical Association 100: 417-21.
  • Smith EH, Whitman RC. 1992. Field Guide to Structural Pests. National Pest Management Association, Dunn Loring, VA.
  • Sonenshine DE, Stout IJ. 1971. Ticks infesting medium-sized wild mammals in two forest localities in Virginia. Journal of Medical Entomology 8: 217-227.
  • Thorner AR, Walker D, Petri WA. 1998. Rocky Mountain Spotted Fever. Clinical Infectious Diseases by the Infectious Diseases Society of America. 27: 1353-1360.
  • U.S. Army Center for Health Promotion and Preventive Medicine (USACHPPM). 2003. Entomological Sciences Program. Aberdeen Proving Ground, MD 21010-5403.

Authors: Wai-Han Chan and Phillip E. Kaufman, University of Florida
Photographs: K. Wilson, J.F. Butler, Lyle J. Buss, University of Florida; Janice Carr, Center for Disease Control
Web Design: Don Wasik, Jane Medley
Publication Number: EENY-443
Publication Date: September 2008. Last revised: January 2013.

An Equal Opportunity Institution
Featured Creatures Editor and Coordinator: Jennifer L. Gillett-Kaufman, University of Florida

entnemdept.ufl.edu

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